The camphor chemotype of Salvia rosmarinus contains 20-45% camphor — markedly higher than other rosemary chemotypes — giving it the most pungent, medicinal aroma and the strongest stimulating and analgesic character of the group. Camphor exerts analgesia through TRPV1 agonism followed by rapid receptor desensitisation, blunting nociceptor signalling; this mechanism was characterised in a landmark study showing camphor activates and then strongly desensitises TRPV1 channels in a vanilloid-independent manner.^[1] Because camphor readily crosses the blood-brain barrier and can act as a convulsant, this chemotype is contraindicated in epilepsy and pregnancy — a risk profile documented in a systematic review of essential oils and seizure events that specifically identified camphor-rich oils as proconvulsant.^[2]